whats up guys :)

 i want to talk about OXY10 that are proven scientifically

to clear pimples fast.

actually,it was packed with white tube with pink or red words of 'oxy' and black work '10'

it contains 10% of Benzoyl Peroxide which medicate the acne pimple

it can kills acne bacteria with effective

get rid off your acne today!

ACNE (Pimples,Zict)

Acne is a common skin disease that causes pimples. Pimples form when hair follicles under your skin clog up. Most pimples form on the face, neck, back, chest and shoulders. Anyone can get acne, but it is common in teenagers and young adults. It is not serious, but it can cause scars.

No one knows exactly what causes acne. Hormone changes, such as those during the teenage years and pregnancy, probably play a role. There are many myths about what causes acne. Chocolate and greasy foods are often blamed, but there is little evidence that foods have much effect on acne in most people. Another common myth is that dirty skin causes acne; however, blackheads and pimples are not caused by dirt. Stress doesn't cause acne, but stress can make it worse.

If you have acne

• Clean your skin gently
• Always use a cleanser for skin
• Try not to touch your skin
• Avoid the sun

Treatments for acne include medicines and creams.

Treatment For PSORIASIS

Psoriasis Treatment

Currently, there is no cure for psoriasis. However, there are many treatment options that can clear psoriasis for a period of time. Each treatment has advantages and disadvantages, and what works for one patient may not be effective for another. Board-certified dermatologists have the medical training and experience needed to determine the most appropriate treatments for each patient.

DiagnosisThere are several forms of psoriasis, and each form has unique characteristics that allow dermatologists to visually identify psoriasis to determine what type, or types, of psoriasis is present. Sometimes a skin biopsy will be performed to confirm the diagnosis.

To choose the most appropriate treatment method, dermatologists consider several factors:

·    Type of psoriasis

·    Severity (the amount of skin affected)

·    Where psoriasis is located

·    Patient’s age and medical history

·    Effects psoriasis has on patient’s overall physical and emotional
well-being

Types of Treatment
Psoriasis treatments fall into 3 categories:

·    Topical (applied to the skin) – Mild to moderate psoriasis

·    Phototherapy (light, usually ultraviolet, applied to the skin) – Moderate to severe psoriasis

·    Systemic (taken orally or by injection or infusion) – Moderate, severe or disabling psoriasis

While each of these therapies is effective, there are also drawbacks.

Some topicals are messy and may stain clothing and skin. Phototherapy can require 2 to 5 weekly visits to a dermatologist’s office or psoriasis clinic for several weeks. Many of the systemic medications have serious side effects and must be combined or rotated with other therapies to maximize effectiveness and minimize side effects. Research is being conducted to find therapies that provide safe, effective, easy-to-use treatment options that provide long-term relief.

Nature Kills Psoriasis

At a health spa in Turkey, a so-called 'doctor fish' is used to treat certain skin diseases. Measuring just a few centimeters the Garra Rufa fish feed on patches of skin affected by psoriasis.

What is Psoriasis?

Psoriasis (sore-EYE-ah-sis) is a medical condition that occurs when skin cells grow too quickly. Faulty signals in the immune system cause new skin cells to form in days rather than weeks. The body does not shed these excess skin cells, so the cells pile up on the surface of the skin and lesions form.

What are the signs and symptoms?
The lesions vary in appearance with the type of psoriasis. There are five types of psoriasis: Plaque, guttate, pustular, inverse, and erythrodermic. About 80% of people living with psoriasis have plaque (plak) psoriasis, also called “psoriasis vulgaris.” Plaque psoriasis causes patches of thick, scaly skin that may be white, silvery, or red. Called plaques (plax), these patches can develop anywhere on the skin. The most common areas to find plaques are the elbows, knees, lower back, and scalp.

Psoriasis also can affect the nails. About 50% of people who develop psoriasis see changes in their fingernails and/or toenails. If the nails begin to pull away from the nail bed or develop pitting, ridges, or a yellowish-orange color, this could be a sign of psoriatic (sore-EE-at-ic) arthritis. Without treatment, psoriatic arthritis can progress and become debilitating. It is important to see a dermatologist if nail changes begin or joint pain develops. Early treatment can prevent joint deterioration.

What causes psoriasis?Psoriasis is not contagious. You cannot get psoriasis from touching someone who has psoriasis, swimming in the same pool, or even intimate contact. Psoriasis is much more complex.

So complex, in fact, scientists are still studying what happens when psoriasis develops. We know that the person’s immune system and genes play key roles. In studying the immune system, scientists discovered that when a person has psoriasis, the T cells (a type of white blood cell that fights unwanted invaders such as bacteria and viruses) mistakenly trigger a reaction in the skin cells. This is why you may hear psoriasis referred to as a “T cell-mediated disease.”

This reaction activates a series of events, causing new skin cells to form in days rather than weeks. The reason T cells trigger this reaction seems to lie in our DNA. People who develop psoriasis inherit genes that cause psoriasis. Unlike some autoimmune conditions, it appears that many genes are involved in psoriasis.

Scientists are still trying to identify all of the genes involved. One of the genes that has been identified is called PSORS1 (SORE-ESS-1). This is one of several genes that regulates how the immune system fights infection.

Scientists also have learned that not everyone who inherits genes for psoriasis gets psoriasis. For psoriasis to appear, it seems that a person must inherit the “right” mix of genes and be exposed to a trigger. Some common triggers are a stressful life event, skin injury, and having strep throat. Many people say that that their psoriasis first appeared after experiencing one of these. Triggers are not universal. What triggers psoriasis in one person may not cause psoriasis to develop in another.

Who gets psoriasis?People worldwide develop psoriasis. In the United States, nearly 7.5 million people have psoriasis and about 150,000 new cases are diagnosed each year. Studies indicate that psoriasis develops about equally in males and females. Research also shows that Caucasians develop psoriasis more frequently than other races. A study conducted in the United States found the prevalence was 2.5% in Caucasians and 1.3% in African Americans.

A family history of psoriasis seems to increase the risk of developing psoriasis. It is important to know that a family history of psoriasis does not guarantee that someone will develop psoriasis.

When do people get psoriasis?Psoriasis can begin at any age, from infancy through the golden years. There are, however, times when psoriasis is most likely to develop. Most people first see psoriasis between 15 and 30 years of age. About 75% develop psoriasis before they turn 40. Another common time for psoriasis to begin is between 50 and 60 years of age.

Does psoriasis affect quality of life?For some people, psoriasis is a nuisance. Others find that psoriasis affects every aspect of their daily life. The unpredictable nature of psoriasis may be the reason. Psoriasis is a chronic (lifelong) medical condition. Some people have frequent flare-ups that occur weekly or monthly. Others have occasional flare-ups.

When psoriasis flares, it can cause severe itching and pain. Sometimes the skin cracks and bleeds. When trying to sleep, cracking and bleeding skin can wake a person frequently and cause sleep deprivation. A lack of sleep can make it difficult to focus at school or work. Sometimes a flare-up requires a visit to a dermatologist for additional treatment. Time must be taken from school or work to visit the doctor and get treatment.

These cycles of flare-ups and remissions often lead to feelings of sadness, despair, guilt and anger as well as low self-esteem. Depression is higher in people who have psoriasis than in the general population. Feelings of embarrassment also are common.

Knowledge is powerAs psoriasis is a life-long condition, it is important to take an active role in managing it. Learning more about psoriasis, seeing a dermatologist to discuss treatment options, and developing a healthy lifestyle can help people live life to the fullest.

Xemodan - The ointment to cure Vitiligo

it is the best thing against vitiligo, much better than melagenina.
But u also can use Tolenorm Ointment which is proven scientifically

TINEA VERSICOLOR ( SKIN FUNGUS)

Tinea Versicolor, also known as pityriasis versicolor is a skin infection caused by fungus. It is found mostly in young people. Any person infected with this will have discolored patches on his skin. These patches can be light or reddish-brown in colour. It is neither a permanent nor a serious infection. Tinea versicolor mainly affects skin on the chest and back. It affects only the top layer of the skin. It is a non-contagious disease. The tinea versicolor rash generally appears on the chest and back and is more common in adolescents than in adults. The fungus that causes the infection feeds on the natural oils that are found on the surface of the skin and so it is theorized that people with an oily skin have a higher risk of suffering from tinea versicolor as compared to those with a dry skin.

The most common symptoms of tinea versicolor are light brown or white patches on the skin. 

These patches are most noticeable during summer season. Rashes on the trunk are one of the symptoms. Generally rashes of tinea versicolor do not appear on the face. Itch on the patches, which become more intense with sweat or overheating. The rash caused by the tinea versicolor fungus can sometimes be mistaken for other skin conditions and rashes such as vitiligo. 

A tinea versicolor infection generally causes a distinctive pattern with larger patches of discoloration surrounded by smaller spots. An individual with tinea white spots may also experience slight scaling of the affected area but this will produce a very fine powdery dry skin layer and not thick scales as is common with other skin infections. Tinea versicolor itching is uncommon in mild cases but may be present in moderate to severe cases. Another peculiar feature of the tinea or pityriasis versicolor rash is that the affected patches are most prominent when the skin is slightly hotter than usual. 

Thus, it is very easy to spot tinea versicolor patches if an individual has been exercising or has been in for a hot shower. This change in color is most noticeable in people who have darkened patches of affected skin as compared to those with lighter patches. The border of the affected patches is very easy to distinguish and is the same color as the rest of the patch. This is another important aspect of the tinea versicolor yeast rash as most other skin conditions cause rashes that have darkened edges. Tinea versicolor and ringworm are often mistaken for each other and this can affect the results of the prescribed treatment. The dermatologist may recommend a KOH Preparation which is a test that is used to determine the presence of a fungal infection and to isolate and establish the exact type of fungal infection.

Treatment for tinea versicolor depends upon the overall health, age and medical history of the infected person. Since tinea versicolor is caused by fungus, you can apply OTC antifungal creams twice daily. Apply antidandruff shampoo that contains selenium sulfide on the patches. Take bath daily, this will remove dead cells and fungus from your skin. Make sure that you use a soft scrubber or a loofa as this will help to get rid of the upper layer of dead skin which contains the fungus. You could have a shower with lukewarm water or slightly warm water but avoid bathing with hot water. Hot water showers tend to strip the skin of all its natural oils and this causes the sebaceous glands in your skin to produce excess sebum. Ketaconazole is a very effective treatment for tinea versicolor and so you could use a shampoo containing this fungicide to help prevent the spread of the skin discoloration. You could also use a tinea versicolor cream or medicated lotion to decrease recovery time.

There are also several tinea versicolor natural treatments that you can prepare at home as these home remedies for tinea versicolor are extremely gentle and will also help to rejuvenate your skin. It is advisable that you follow a regular skin care schedule that includes several homemade remedies for tinea versicolor. You can make a homemade body scrub for tinea versicolor by lightly grinding a handful of unpolished rice so that you obtain a coarse flour. You can add a few spoons of fresh tomato juice to this flour until it forms a paste. Shower as usual and then apply this paste all over the affected areas of skin. Allow the paste too remain on your skin for about 5 minutes and then gently massage it into your skin. The tomato juice is acidic in nature and this prevents the spread of the fungus. The rice flour contains several nutrients that are essential for skin health and since it is also coarse it serves as an exfoliating agent. You may also use apple cider vinegar for tinea versicolor as vinegar is acidic in nature and provides a hostile environment for fungus and thus helps to control the spread of the infection. When treating tinea versicolor with home remedies, it is important that you keep in mind that it may take a while for your skin top regain its original color.

TREATMENT FOR TINEA VERSICOLOR

Tea tree oil is a very potent natural fungicide and there are several tinea versicolor tea tree oil remedies that you can use to treat this condition. It is important to dilute the tea tree oil with a carrier oil before you use it as tea tree oil is an essential oil and contains very strong natural compounds. Mix equal quantities of tea tree oil, olive oil, and sweet almond oil and use this blend of oils after your shower instead of a moisturizing cream.

"" I have been working on a remedy for over 1 1/2 years! I finally have come up with the right combination to battle fungal infections of the skin.

Tinea Versicolor is a skin fungus that causes the affected skin to change color and become either lighter or darker than surrounding skin.

My daughter who is extremely fair skinned has had this condition for several years. Prescription ointments and creams as well as a prescription anti fungal pill did not work for her in fighting this fungal infection.

Since we believe natural is healthier and effective We have been concocting various oils, lotions, and balms over the past year to fight this fungal infection of the skin.

I have come up with a pomade that has been effective!
My daughter has been applying this pomade for a couple of months now and it is making progress in ridding her skin of this fungus.

I have used the essential oils of lavender, eucalyptus, menthol,neem,cedarwood, peppermint, oregano, and thyme. Blended into my base of organic castor oil,Ingredients: Castor oil, organic calendula oil,carnuba wax,Ricinus Communis (Castor) Seed Oil (and) Hydrogenated Castor Oil (and) Beeswax (and) Copernicia Cerifera (Carnauba) Wax.

Apply this twice daily to skin that is infected with fungus.
This is a 1 ounce container. Convo me for larger size ""

Selenium Sulfide has been proven to be very effective in the treatment of tinea versicolor and so there are several tinea versicolor remedies that involve the use of Selenium Sulfide shampoos. The Tinea versicolor selsun blue remedy is one of the most commonly used tinea versicolor treatments and when used for a prolonged period has remarkable effects. To use this remedy, you will need to stand under a warm shower for a few minutes and then lather your body with this shampoo. You would need to leave the lather on for 5 minutes or so and then use a loofa to gently exfoliate your skin. You can repeat this procedure once more on the affected areas of your skin. This tinea versicolor selsun blue remedy will help to treat the condition as well as prevent it from spreading. You can also continue to use this remedy twice a month for the next few months as it is also an excellent preventative method and will prevent the recurrence of tinea versicolor. Even if you know how to treat tinea versicolor, it is best that you speak to a dermatologist before your decide on a treatment method. Tinea versicolor on the face is very rare but when it occurs it can be extremely prominent as facial skin tans easily. When you have tinea versicolor, tanning in any form should be avoided.


Sunbathing at the beach and tanning beds will only make the discoloration prominent. There has been a lot of research towards finding out if tinea versicolor is contagious and unfortunately there is no definite consensus. Since the fungus that causes tinea versicolor is naturally present on our skin, it would obviously follow that it wouldn’t really be considered contagious. However, some studies have found that people who frequently share personal items like clothing have a higher risk of contracting this skin condition. So although tinea versicolor is not considered to be a contagious skin condition, it would be best to avoid sharing personal items as far as possible.

BASAL CELL CARCINOMA

Basal cell carcinoma (BCC) is type of skin cancer that found the most in human. There are about one million American diagnosed and an estimated 20,000 people who die from skin cancer or basal cell carcinoma worldwide each year.

Basal cell carcinoma mostly appears on the face, ears shoulder and neck where the skin is easily exposed to sunlight or dust, X –ray treatment, low vitamins intake and some chemical may cause serious BCC. However, it can also appear on other parts of body such as the abdomen, leg, and scalp exposed to sun’s ultraviolet rays. Early diagnostic and treatment will help to reduce damage the skin and cause an ulcer known as a rodent ulcer. In some cases, contact with arsenic, exposure to radiation, open sores, lesions that do not heal, inflammatory skin conditions, and complications of burn scars, infection, vaccination, or even tattoos are contributing factors

Basal Cell Carcinoma Causes

-          There is some people develops basal cell carcinomas by exposed to fiberglass dust and dry cleaning. Also, prior non-diagnostic X-ray treatment for skin conditions increased risk of both cancers.

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-          Several studies have shown an association between cumulative ultraviolet exposure and risk of basal cell carcinoma, although the magnitude of risk conferred has been small.

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-          There is also other non-ultraviolet environmental exposures that have been associated with increased risk of basal cell carcinoma include ionizing radiation, high dietary energy (especially fat), low intake of vitamins, and various chemicals and dust. Exposure to arsenic predisposes to multiple basal cell carcinomas.

-          Lately UK has also been reported that 2% of such tumors could be associated with exposure to radon.

-          Some research found squamous cell carcinomas in people exposed to insecticides, herbicides, fungicides and seed treatments as well petroleum products, grease, and several other exposures.

Basal Cell Carcinoma Prevention

-          If you would like to get tan, do so slowly, starting at 15 minutes a day, max. Work up to greater exposure, but only if there is no damage, not even slight damage, to your skin. Basal cell carcinoma tends to be more virulent among people who get most of the tanning from tanning beds.

-          The most important nutrients for preventing basal cell carcinoma are antioxidants supplement and vegetable. The main principal is using n-acetyl cysteine (NAC), selenium, and vitamins C and E. NAC works synergistically with vitamins C and E to protect the “watchdog” gene p53 that keeps skin cells with damaged DNA from continuing to grow and multiply. Selenium and vitamin E help the skin make the “super-antioxidant” glutathione, and another antioxidant, alpha-lipoic acid, will help keep glutathione from being broken down in the liver. Alpha-lipoic acid is especially important for people who work or exercise outdoors.

-          What else can you do to relieve and prevent basal carcinoma? Number one, wear sunscreen or sun block lotion. The recommendation used to be to use at least SPF-15 on sun-exposed skin in the summer. Now more doctors tell their patients to use a stronger (higher number) sun screen any time of year.

Basal Cell Carcinoma Treatment

Treatment of BCC is depending on the size, depth, and location of the basal cell cancer. Here are treatments options that available include:

MOHS surgery

Radiation therapy

Cryosurgery (freezing cancer cells)

Topical chemotherapy

Photodynamic therapy.

Treating Basal cell with MOHS is by far the best way to prevent the Basal cell cancer from coming back (about a one percent chance of returning) while other types of basal cell treatment are less effective (about a ten percent return rate). MOHS is a surgical procedure that involves removing tumor tissue layer by layer. Each of the layers of removed tissue is then scanned for cancer cells under a microscope

SKIN DISEASES: VITILIGO

What is vitiligo, and what causes it?

Vitiligo (pronounced vit-ill-EYE-go) is a pigmentation disorder in which melanocytes (the cells that make pigment) in the skin are destroyed. As a result, white patches appear on the skin in different parts of the body. Similar patches also appear on both the mucous membranes (tissues that line the inside of the mouth and nose) and the retina (inner layer of the eyeball). The hair that grows on areas affected by vitiligo sometimes turns white.

The cause of vitiligo is not known, but doctors and researchers have several different theories. There is strong evidence that people with vitiligo inherit a group of three genes that make them susceptible to depigmentation. The most widely accepted view is that the depigmentation occurs because vitiligo is an autoimmune disease -- a disease in which a person's immune system reacts against the body's own organs or tissues. People's bodies produce proteins called cytokines that, in vitiligo, alter their pigment-producing cells and cause these cells to die. Another theory is that melanocytes destroy themselves. Finally, some people have reported that a single event such as sunburn or emotional distress triggered vitiligo; however, these events have not been scientifically proven as causes of vitiligo.

Who is affected by vitiligo?

About 0.5 to 1 percent of the world's population, or as many as 65 million people, have vitiligo. In the United States, 1 to 2 million people have the disorder. Half the people who have vitiligo develop it before age 20; most develop it before their 40th birthday. The disorder affects both sexes and all races equally; however, it is more noticeable in people with dark skin.

Vitiligo seems to be somewhat more common in people with certain autoimmune diseases, including hyperthyroidism (an overactive thyroid gland), adrenocortical insufficiency (the adrenal gland does not produce enough of the hormone called corticosteroid), alopecia areata (patches of baldness), and pernicious anemia (a low level of red blood cells caused by the failure of the body to absorb vitamin B₁₂). Scientists do not know the reason for the association between vitiligo and these autoimmune diseases. However, most people with vitiligo have no other autoimmune disease.

Vitiligo may also be hereditary; that is, it can run in families. Children whose parents have the disorder are more likely to develop vitiligo. In fact, 30 percent of people with vitiligo have a family member with the disease. However, only 5 to 7 percent of children will get vitiligo even if a parent has it, and most people with vitiligo do not have a family history of the disorder.

This year the San Raffaele Scientific Institute in Milano will host the First World Vitiligo Congress, between 23 - 24 September. This conference will focus on vitiligo cure research.

Anti-Vitiligo has originated in the subcontinent and its success in the brown races has been tested over centuries. Its recent availability across the globe has made it possible to evaluate its healing potential in other races as well, and the data has shown that success of anti-vitiligo is not specific to any race.
Anti-Vitiligo has been found to improve all variants of the vitiligo disorder including generalized vitiligo1 (universal vitiligo, acrofacial vitiligo and vitiligo vulgaris) and localized vitiligo1 (segmental vitiligo and focal vitiligo)


Vitiligo is a disorder characterized by patchy loss of skin pigmentation due to immune attacks on melanocytes, which can be caused by defects in many genes. Variations in genes that are part of the immune system or part of melanocytes have both been associated with vitiligo. The immune system genes are associated with other autoimmune disorders.
In one case, the gene TYR, which makes the melanocyte more susceptible to the immune system in vitiligo, also makes the melanocyte more susceptible to the immune system in the skin cancer malignant melanoma. So people with vitiligo caused by the TYR gene are less likely to have malignant melanoma.
A genomewide association study found 10 independent susceptibility loci for generalized vitiligo, responsible for 7.4% of the genetic risk. Some patients had vitiligo alone; others had generalized vitiligo with other autoimmune diseases. Most loci were associated with both forms. (The exception was PTPN22, which was only associated with generalized vitiligo.) In the MHC region, which controls the immune system, major association signals were identified in the class I gene region (between HLA-A and HLA-HGC9) and class II gene region (between HLA-DRB1 and HLA-DQA1). Outside the MHC region, association signals were identified near RERE, PTPN22, LPP, IL2RA, GZMB, UBASH3A and C1QTNF6 genes, which are associated with other autoimmune diseases. TYR encodes tyrosinase, which is not a component of the immune system, but is an enzyme of the melanocyte that catalyzes melanin biosynthesis, and a major autoantigen in generalized vitiligo. The major alleles of TYR are associated with vitiligo, and the minor alleles are associated with malignant melanoma. Vitiligo-associated 402R tyrosinase may be more efficiently presented to the immune system. Melanoma-associated 402Q may fail to be identified by the immune system.
The transcriptional profile of melanocytes from vitiligo patients have been studied. Oligonucleotide microarrays containing approximately 16,000 unique genes were used to analyse mRNA expression in melanocytes from vitiligo patients and age-matched healthy controls. In total, 859 genes were identified as differentially expressed.
Vitiligo is sometime associated with autoimmune and inflammatory diseases,commonly thyroid overexpression and underexpression. A study comparing 656 people with and without vitiligo in 114 families found several mutations (single-nucleotide polymorphisms) in the NALP1 gene.The NALP1 gene, which is on chromosome 17 located at 17p13, is on a cascade that regulates inflammation and cell death, including myeloid and lymphoid cells, which are white cells that are part of the immune response. NALP1 is expressed at high levels in T cells and Langerhan cells, white blood cells that are involved in skin autoimmunity.
Among the inflammatory products of NALP1 are caspase 1 and caspase 5, which activate the inflammatory cytokine interleukin-1β. Interleukin-1β is expressed at high levels in patients with vitiligo. There are compounds which inhibit caspase and interleukin-1β, and so might be useful drugs for vitiligo and associated autoimmune diseases. In one of the mutations, the amino acid leucine in the NALP1 protein was replaced by histidine (Leu155->His). The original protein and sequence is highly conserved in evolution, and found in humans, chimpanzee, rhesus monkey, and bush baby, which means that it is an important protein and an alteration is likely to be harmful. Addison's disease (typically an autoimmune destruction of the adrenal glands) may cause vitiligo.